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This is a difficult-to-diagnose inflammation derived from the localized release by afferent neurons of inflammatory mediators such as substance P, CGRP, neurokinin A (NKA), nitric oxide, vasoactive intestinal polypeptide, to a lesser extent Serotonin 5-HT (other serotonin neurotransmitters are deficient) and endothelin-3 (ET-3)

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In parallel, the NeuN intensity tended to decrease in SOD1 G93A mice, and KDS12025 potently restored this signal (Fig