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Mechanistic validation was further provided by the WILLOW89 study, which confirmed the compound’s capacity to potently inhibit neutrophil serine protease activity, including neutrophil elastase and proteinase 3, thereby attenuating the protease-antiprotease imbalance and subsequent neutrophilic airway inflammation and parenchymal destruction characteristic of bronchiectasis pathophysiology [75]

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Future research should continue to explore the mechanisms underlying the actions of melatonin and its potential applications in clinical settings