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Not only does excess superoxide itself cause increased oxidative stress, but it can also react with nitric oxide (NO • ) to produce peroxynitrite [18], which, in turn, can oxidize tetrahydrobiopterin (BH 4 ), thus reducing its availability to eNOS [17,18]

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63, 64 In normal physiological circumstances, adipocytes release anti-inflammatory factors such as adiponectin, transforming growth factor-beta, interleukin-10 and nitric oxide, which promote insulin sensitivity and antiatherogenic effects

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