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The maintenance of glutathione cycle and survivability in neurotoxin exposed cells by Sp, led us to test its plausibility in recovering the degenerative phenotypes of well-established Drosophila Parkinson mutant model park 13

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We reported that chronic alcohol exposure reduced K Ca 2.2 channel surface expression and increased the synaptic expression of GluN1 and GluN2B subunits of the NMDA receptor [91]