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The possible explanation for the increased spontaneous acrosome reaction as seen in this study could be due to inhibition of PKA, actin polymerisation or CAMKII and PLD pathways, which needs to be further interrogated

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Similarly, lactylation of ALDOA at lysine 230/322 in liver cancer stem cells (LCSCs) facilitates the dissociation of ALDOA from DEAD-box helicase 17, ultimately enhancing glycolysis, proliferation, and migration of liver cancer