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At this stage, high HO-1 expression may disrupt iron ion homeostasis, leading to elevated free iron levels in vascular cells ( BACH1 and promote the translocation of HO-1 from the cytoplasm to the mitochondria, leading to mitochondrial iron overload and the accumulation of lipid ROS ( Based on current research, the mechanistic interplay between HO-1 and oxidative stress has been partially elucidated

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Notably, the WHI randomized trials (163, 168) and a meta-analysis (169) have demonstrated that treatment with CEE alone significantly decreases the risk of breast cancer