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Although endophilin binding to VGLUT1 reportedly facilitates endocytosis of VGLUT1 during mild prolonged repetitive stimulation (Voglmaier et al., 2006) and reduces SV-release probability (Weston et al., 2011), a recent study using hippocampal neurons suggests that it also reduces SV mobility mediated by an additional endocytic protein, intersectin (Zhang et al., 2019), presumably representing a molecular mechanism underlying faster recovery after STD in the absence of VGLUT1

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