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The following sections summarizes (a) the role of oxidative stress in initiation of AKI and their transition to fibrotic kidney that progresses ultimately to chronic kidney disease, (b) dysregulation of molecular signaling pathways associated with initiation and transition of AKI to CKD, (c) current therapeutic approaches for the treatment of kidney disease, and (d) promise and potential of target molecules dysregulated by oxidative stress during development of kidney disease

Kirkastava

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