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First, enriched with Th1-like signatures in CD4+ T cells, M1-like pro-inflammatory signatures in monocytes, cytotoxic effector signatures in CD8+, T cells and NK cells, and memory signatures in B cells

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Similar content being viewed by others Introduction Sickle cell disease (SCD) is an inherited hematological disorder caused by an A to T mutation in the adult β-globin gene with the product of hemoglobin S which polymerizes under hypoxia conditions 1,2

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