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N‐acetylcysteine (NAC) in neurological disorders: mechanisms of action and therapeutic opportunities - Bavarsad Shahripour - 2014 - Brain and Behavior - Wiley Online Library

This inflammatory form of cell death disrupts osteoblast and macrophage function in bone, damages intestinal epithelium, and maintains chronic microglial activation in the CNS, resulting in persistent inflammation [48]

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Capsules:Unflavored Powder (30 Servings)

In addition, GSH has been shown to regulate nitric oxide homeostasis (Hogg, 2002), modulate the activity of proteins by post-translational modification (protein S-glutathionylation) (Pompella et al., 2003), and modulate the activity of neurotransmitter receptors (Oja et al., 2000)