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However, enhancing lactate levels or inhibiting MCT4, a pivotal lactate transporter, promotes α-MHC-K1897la and α-MHC-Titin interaction, alleviating heart failure

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falciparum parasites with deletion of the oxoglutarate dehydrogenase (ΔKDH), which would force malate to enter the mitochondria to supply oxaloacetate via MQO and oxoglutarate to exit from the organelle ( From the above, it follows that a constant supply of oxaloacetate for citrate synthase would require an efficient MQO