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Its pathogenesis is associated with alterations in the innate immune system and neurovascular dysregulation [42]

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Figure 1 3 ROS-mediated signaling pathway The physiological consequences of oxidative stress primarily stem from its capacity to modulate diverse signaling pathways, including the nuclear factor κB (NF-κB), mitogen-activated protein kinase (MAPK), Kelch-like ECH-associated protein 1/nuclear factor E2-related factor 2 (Keap1/Nrf2), and AMP-activated protein kinase (AMPK) pathways

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