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Researchers suggest that the GHK sequence may occur endogenously, specifically being released by cells, including fibroblasts, macrophages, and lymphocytes, during damage, as a result of collagen breakdown

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One example in diabetic cohorts treated with MitoQ, the clinical protocol was designed combining DM endpoints (HbA1c and endothelial function) in a more preferable way than isolating redox-specific effects from glucose-lowering biomarkers

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