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Testing this hypothesis will require direct investigation in established toxic (e.g., cuprizone) and autoimmune (e.g., EAE) models of demyelination, which represents a critical next step for the field as summarized in Figure 1

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Hyperuricemia, in association with higher mortality rates in women with CKD, has been shown to act as a stimulating factor to individuals at risk of CHD related-events [79]