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It has been shown that germline mutations in RAS proteins or in RAS regulators, such as NF1, PTPN11 , or SOS1 , lead to several congenital developmental disorders, such as neurofibromatosis type 1, Noonan, or Costello syndromes, respectively ( Numerous mechanistic studies from the last two decades support the notion that each RAS protein displays specific downstream signaling ( Early evidences from plasma membrane perturbation studies support the idea of compartmentalized RAS protein signaling