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We understand this is further evidence that myelin inhibitory proteins, such as Nogo-A, may serve as a converging point between inflammation, synaptopathy and glial cells in demyelinating diseases, as is described in Figure 3

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Overall, the data to date indicates that 4-1BB co-stimulation skews CAR T cell metabolism towards OXPHOS and FAO and promotes a less differentiated phenotype (with a higher replicative potential)